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Interferons, mediators of the host defence against viral infection and carcinogens, include type 1 interferon (IFN-alpha/beta) and type 2 interferon (IFN-gamma). The four genes encoding IFNs are differentially regulated in response to stimuli and the genes can be induced in a cell-specific manner. Recent progress has centred on the identification and structural characterisation of IFN-alpha, IFN-beta and IFN-gamma. Each is encoded by a family of gene-products, each of which is processed to a single, 20-kDa protein. IFNs inhibit virus replication by various mechanisms: they modulate the viral life cycle or the induction of antiviral proteins, they promote the maturation of dendritic cells and they augment the activity of natural killer cells. Other biological activities include induction of the transcription of certain genes and modulation of cell proliferation, differentiation and immunoglobulin gene rearrangement. The therapeutic utility




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